End results of Acute Inflammation::
End results of acute inflammation mainly depend upon the resistance of the patient and /or virulence of the causative agent if the resistance power of the patient is good and virulence of the causative agent is ineffective to produce necrosis (cell-death) then exudate may be completely reabsorbed by lymphatics and inturn it may re-enter in to the blood vessels if the necrosis is present the dead tissue in inflammed area undergoes softening and liquifection this phenomenon is known as suppuration this finally results in pus formation if inflammation exudate fails to undergo resolution it is converted into granular tissue which heals by fibrosis and finally there is formation of scar
If the causative agent is highly virulent and /or the resistance of the patient is poor then the inflammation becomes sub-acute or chronic and lasts longer
Tissue-repair::
It is the process of the reconstruction of the part to a more or less similar structures it begins during the active phase of inflammation but can be completed only after all harmful substances have been neutralized or removed from the site of injury New cells originate by cell duplication from the stroma (i.e. the supporting connective tissue) or from the parenchyma (cells which form part of the organ) if parenchymal cells are involved in tissue repair a perfect or nearly perfect reconstruction of the injured tissue may occur however if the fibroblasts of stroma are involved there is fibrosis resulting in scar formation
Protein rich diet vitamin A vitamin B (thiamine nicotinic acid riboflavin ) and Vitamin c are all necessary and hasten -up the healing processes Vit D helps in healing of fractures
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