MOLYBDENUM(MO):::
It is transition metal that forms oxides and is a component of pterin coenzyme essential for the activity of xanthine oxidase sulfite oxidase and aldehyde oxidase Genetically conditioned sulfite oxidase deficiency was described in 1967 in a child with mental retardation convulsions opisthotonus and lens dislocation this disorder was due to the child s inability to form the molybednum coenzyme despite the presencefo adequate molybdenum
Sulfite toxicity due to molybdenum deficiency was noted in a patient on long term total parenteral nutrition (TPN) who developed tachycardia tachypnea headache nausea vomiting and coma A metabolic study showed high levels of sulfite and xanthien and low levels of sulfate and uric acid hin his blood and urine which led to the diagonosis giving ammonium molybdate 300 ug /day IV led to a dramatic recovery both genetically conditioned and nutritional deficiencies of molybdenum are rare the intake of molybdenum varies from 100 to 500 ug/day and is derived principally from organ meats whole grain cereals and legumes
The food and nutrition board states that a safe adequate intake of molybdenum is 75 to 250 ug/day for adults and 25 to 75 ug/day for children aged 1 to 6
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